All of Tornus's Comments + Replies

Tornus10

This stuff is super hard.

I'd recommend (with reservations) Consent Academy, who do a lot of training on incident response, accountability processes, etc. They're good folks who have figured out a lot of really useful things about doing this kind of work.

Their classes can sometimes get pretty rambling and theoretical, but I've learned a lot from them.

Tornus30

Strong work: thank you!

I believe there's a small mistake: in the first table (after "In equilibrium, we see the following amounts of sars-cov-2 relative to no filtration:"), I believe the second column should be labeled "presence", not "reduction".

2jefftk
Thanks; fixed!
Tornus30

Lots of cool data here—thank you!

(Edited to remove a comment based on misremembering the Most Penetrating Particle Size)

Tornus30

I'm overdue for making another pass through the latest data, so my opinions on this are weakly held. But briefly: my current thinking is that many people (including Zvi and me) have made the mistake of conflating a number of different phenomena into the single category of "long covid". I believe Zvi is correct that if a large number of people were suffering long-term debilitating impact, we'd know it.

I suspect that after I plow through the data again, I'll update significantly in the direction of believing that:

  • "Long covid" is a debilitating phenomenon t
... (read more)
Tornus20

Eliezer, back in 2009:

Yet there is, I think, more absent than present in this "art of rationality"—defeating akrasia and coordinating groups are two of the deficits I feel most keenly.

This is not a small project, and I'm too new here to have a clear sense of how it might happen. But this feels important.

Answer by Tornus90

To more directly address your initial question: to my mind, Zvi's analysis isn't obviously wrong, but it's pretty far to the optimistic end of what I see as the reasonable range.

My best model suggests that for me (55 but very healthy), 1,000 µCoV of risk has an expected life cost of about 15 minutes.

Based on that, my approach to risk is very situational. Is eating in a restaurant worth 75 minutes of lying in bed with flu wishing I was dead (based on today's numbers)? No, it isn't. Is going to a friend's wedding worth that? Yes, it probably is.

1Sameerishere
Thanks! I'm curious what you think of his argument that if debilitating long COVID were common, that would be obvious anecdotally and in aggregate statistics:
9Richard_Kennaway
I am amused to note that some people do think that going out and drinking to excess with friends is worth 75 minutes of lying in bed with a hangover wishing they were dead.
Answer by Tornus50

I'd love to see a more structured approach to the kinds of questions you're raising here. LW does a good job of creating a space for smart people to share their thoughts about individual topics, but isn't so good at building toward a coherent synthesis of all those pieces.

The original microCOVID white paper did a good job of summarizing a lot of relevant evidence back in the day, but (like the rest of the site) has been only sporadically updated.

Put me down as tentatively interested in being part of some larger project, if one comes together.

Also: may I humbly request that if this ever takes off, it be named LessSick?

5Michael K
This feels like a hole in LessWrong in general that would seem to be worth addressing. Maybe a general initiative for synthesis on various topics should be created, not just about COVID?
Tornus10

That all makes complete sense.

And yes, the specifics of the population make a huge difference. Honestly, I think that accounts for the breadth of my estimate range more than uncertainty about abstract test performance does.

Tornus10

I think it's important to emphasize that antigen+ people are much more contagious than antigen-. It's hard to quantify that, but based on typical differences in Ct value, it's probably a very substantial difference (factor of 10+?).

You're absolutely right that the reference class is the key issue (if there's one thing I've learned from hanging out with epidemiologists, it's that they're always grumpy about people using the wrong denominator).

In a perfect world, where everyone with any symptoms whatsoever stayed home and was scrupulous about following what ... (read more)

Tornus10

Let me give you two answers for the price of one:

  1. FDA and others have been very clear about this: you should use the tests as directed.

  2. I (a decades-long amateur epidemiologist who's done a deep dive on antigen test research), my partner (a medical epidemiologist who works full-time on Covid), and several other epidemiologists I'm aware of, all use throat + nasal swabs.

I wouldn't worry at all about false positives: they really haven't been an issue with antigen tests. If I got a positive from a throat + nasal swab, I'd follow it up with a nasal-only... (read more)

Tornus20

Why do I respect Michael Mina? Weak but deep answer: because in my experience he’s been consistently smart, and insightful about Covid and especially about testing (and he’s a professional epidemiologist / immunologist). Strong but shallow answer: because my partner, who is a medical epidemiologist working full-time on Covid, thinks highly of him.

If you’re not already familiar with her, you might also be interested in Katelyn Jetelina (Your Local Epidemiologist). IMHO, she produces by far the best deep research summaries for laypeople. Here’s a recent piec... (read more)

5Connor_Flexman
I think my current expectation of risk reduction from antigen tests is more like 20-60% than <10%, but I'll also note that it matters a lot what your population is. In Elizabeth's social circle my guess is that most people aren't coming to parties if they've had any suspected positive contact, have any weak symptoms, etc, such that there's a strong selection effect screening out the clearly-positive people. (Or like, imagine everyone with these risk factors takes an antigen test anyways—then requiring tests doesn't add anything.) I haven't read this whole thread but for the record, I often agree with Michael Mina and think he does great original thinking about these topics, yet think in this case he's just wrong with his extremely high estimates of antigen test sensitivity during contagion. I think his model on antigen tests specifically is theoretically great and a good extrapolation from a few decent assumptions, but just doesn't match what we see on the ground. For example, I've written before about how even PCRs seem to have 5-10% FNR in the hospitalized, and how PCR tests look even worse from anecdata. Antigen tests get baselined against PCR so will be at least this bad. We also see things like a clinical trial on QuickVue tests that shows only ~83% sensitivity. Admittedly other studies of antigen tests show ~98% sensitivity, but I think publication bias and results-desirability bias here means that if the clinical trial only shows 83%, then that's decent evidence that studies finding higher are a bit flawed. I would not have guess they could get to 98% though so there's something that doesn't make sense here. I know the standard heuristic is to trust scientific findings over anecdata, but I think in this case that should be reversed if you're extremely scientifically literate and closely tracking things on the ground. Knowing all the things that can go wrong with even very careful scientific findings, I just don't trust these studies claiming very high sen
2Elizabeth
Apologies for poor formatting, I'm on mobile. It looks to me like the first study Jetelina cites agreed with me? Average time to positive antigen test was 3 days after positive PCR. Everyone developed symptoms within 2 days of the positive PCR (which is surprising in and of itself), and at least 4/30 people spread covid before getting a positive antigen test, which presumably would have been higher if they didn't have the PCR to warn them. PCR is more sensitive than cue tests so this doesn't translate directly, but I consider it to bolster the case against preemptive antigen testing being useful. In the second study, which was on demand not preemptive testing, antigen tests detected 50% of PCR positives. If I understand your claim correctly, it's that the PCR+/antigen- people aren't very contagious? I agree that antigen+ people are on average more contagious, but since symptomatic people (should) stay home, I don't think that's the right reference class. The meaningful work is done identifying people who couldn't otherwise be identified as infectious.
Tornus60

I'm wondering if you can explain a bit more about your thinking here.

From my perspective, there's a strong prior that antigen tests work well for Covid screening:

  1. There are numerous peer-reviewed studies to that effect. Here are two recent ones, but there are many others Soni et. al., Jüni et. al..

  2. Multiple experts in the field continue to assert that antigen tests work very well for Covid screening. Michael Mina is extremely knowledgeable on this topic and particularly vocal about it.

It's important to note here that PCR / antigen discordance early ... (read more)

2Elizabeth
The Soni study shows only 20% of cases were antigen positive the same day they became PCR positive (and because they were sampled only every 48 hours, the actual power is less still). The second showed that antigen tests didn't begin to pick go positive until 2 days after the positive PCR test.  So I don't see how these contradict me. I don't have high expectations for people marketing themselves as covid experts. Maybe this particular guy is good, which would be great for me because doing this research myself is time consuming. What makes you value this guy in particular? There is a more complicated argument that the genetic tests are picking up infected but uninfectious people. That's certainly possible.  I've got my stack of anecdotes about people who caught covid from someone with no or ambiguous symptoms and multiple negative tests, but those don't have a denominator either. However given the primate challenge data that current vaccines don't affect nasal viral titers or immune response (even while they're providing lots of protection to the throat and lungs), I'm confused by this.   This particular data is for INO-4800, but unless something very weird is happening the same principle would apply to the intramuscular mRNA vaccines. Depends what your starting position was. I'm arguing against treating antigen tests as near-100% screening that removes the need for other safety measures, which seems to be the attitude of the party hosts I'm seeing. If you already viewed them as "better than nothing but not by much", I don't think you need to change at all. 
Tornus90

This is a really good start, and I look forward to the inevitable improvements in the quality of discourse. But to fully leverage the potential of this exciting new system, I think you should create a futures market so we can bet on (or against) specific individuals writing good posts in future.

Also: from now on, I vow to ignore any and all ideas that aren't supported by next-level puns.

Tornus40

When I had an acute bout of insomnia, one of the things I found most helpful was listening to sleep-focused bedtime stories. The key part wasn't the sleepy imagery, but rather just having something boring and inconsequential that my mind could latch onto, to replace the busy inner dialog.

I particularly like the stories from the Headspace app—they're slightly randomized each night, which prevents you from using the story progress as a timer ("Oh no, we're up to the skunk already and I'm still not asleep!")

Related: I also found it extremely helpful to get ri... (read more)

Tornus50

I'm excited about this sequence, and look forward to the rest of it.

Having just read this introduction, it almost feels to me like the tail is wagging the dog. I completely agree that relinquishing options is a critically important part of civility. But my instinct is that the relinquishment is in service of a greater (and defining) goal, not the goal itself. So, something like "civility is prioritizing cooperation over autonomy, which in many cases requires relinquishing physically possible options". But I assume it will all become clear as the sequence proceeds.

Tornus10

Thank you for this.

I want to start by echoing your gratitude to the microCOVID team: they've done amazing work and the tool they've produced has been incredibly valuable to me and to many others. And I agree with your assessment that microCOVID is much less useful than it has been in the past. I'll add to your points:

  • Their calculation of prevalence strikes me as far too clever. I understand what they're trying to do, and it makes sense in theory. But Covid surveillance is something I know a lot about, and I believe they're over-driving the data. Test pos
... (read more)
Tornus290

Thank you for the reminder to explain and not scold—I shall strive to do so.

4Alexander Gietelink Oldenziel
No. Thank you for exemplifying Rationalist virtue!
Answer by Tornus-30

I'd caution you against spending too much time diving down infinite crank rabbit holes: true believers will always find some new detail or theory for you to rebut. At some point, if someone is committed to denying the clear scientific consensus, there's no point trying to get through to them.

At a high level, we have a pretty deep understanding of how covid vaccines work and how they perform over time, and there's absolutely nothing in there to suggest that, unlike every other vaccine ever, covid vaccines display the bizarre transition from positive protect... (read more)

6abstractapplic
mRNA vaccines are a new invention, so that line of reasoning isn't particularly reassuring. An extremely helpful paragraph, which taught me several things I'm embarrassed not to have known; that's worth a strong upvote by itself. In retrospect, I can see how I gave the impression that ADE was the main concern, instead of just the worst-case scenario; that's my bad. The scenario that seems not-vanishingly-unlikely to me is unprecedentedly homogenous immune responses incentivizing unprecedentedly rapid antigenic drift, until patients end up with immune responses (enshrined by antigenic sin) less effective than what their bodies would have come up with by themselves. I'll edit the OP to clarify. Thank you for expaining the shift in protection, and for getting me to make the OP clearer. You'll definitely get to allocate part of the $2k; I'll figure out how much after more people have had a chance to provide answers.

abstractapplic did their best in communicating fairly and honestly their epistemic state - considering the fact that Covid vaccination has become a highly politically charged topic they did a commendable thing by doing their utmost to be transparent about their motives and their uncertainty.

yes usually blindly following mainstream advice is better than following cranks and sometimes one can waste a lot of time debunking cranks - but I hope you'll agree that mainstream advice is not always the best. I do agree about being aware of the danger infinite crank ... (read more)

Tornus10

To a greater or lesser extent, I think that's true for many of us here. Which is a good thing in some ways, but can make it challenging to fully understand and engage with people who are more hive-oriented.

Tornus50

The key thing is that it's low-commitment / low-guilt. I was inspired to start it by a friend who started a book club during the pandemic, fell catastrophically behind on the reading, and ultimately ended up ghosting her own book club.

I've noticed that book clubs tend to become machines for making people feel guilty / overloaded, so I tried hard to avoid that. We do a book every 2 - 3 months, and the default expectation is that people won't attend unless that specific book is interesting to them.

Shortly before the discussion, I send out a summary of the bo... (read more)

Tornus50

As a side note, I run a thing that's like a book club but different and we're talking about The Righteous Mind on Saturday 1/22. We have room for a few more thoughtful people—feel free to message me if you're interested.

1oge
How is your book club different?
Tornus80

Vaccine-required zones seem unworkable to me: ours is a highly connected society and it's common for a single household to have members who have jobs / school separated by many miles. Self-sufficiency is completely impossible in the modern world—the closest example is probably North Korea, but that's probably not a model we want to pursue.

There are also immense transaction costs here: there's no area where everyone wants (or doesn't want) to be vaccinated, so implementing this would require massive migration, with immense costs.

It seems to me you've hit on... (read more)

1MikkW
In the post I say: If you have a community of vaccinated people, the nice thing is that even if some of them have jobs that put them around non-vaccinated people, first: that person is less likely to get infected at any given point in time, and second: if/when they do get infected, the people they are in contact with will be much less likely to get infected as a result. Both of these combine to mean that someone who lives in a vaccine-only zone will be better-protected than someone who lives outside of one. I agree that there transaction costs involved, since moving is not a small deal. This can certainly slow down the proposal relative to the ideal, but it hardly seems like a killer argument against implementing vaccine-only zones (as long as it is ensured that implementing the zone doesn't require forcing any existing population to move, i.e. founding in a currently sparsely populated area)
Answer by Tornus70

I'm afraid I only have time for a short, partial response today. Short version: Covid surveillance is hard, and there's lots of noise in the data. But there are lots of smart people working hard on this, and in the aggregate we actually have a pretty good idea what's going on.

I'll address one of the questions you asked specifically:

So where are these numbers for variant spread coming from? Maybe hospitals do have special genetic tests and reliably do those? But then isn't there going to be a pretty strong bias based on the fact that these are only for peop... (read more)

-3Valentine
Thank you. This is clear and points me in directions that let me explore more and see through the fog of war.
2lsusr
I live near the UW. As far as I can tell, the UW has done a great job of pandemic response. I got a COVID test from them early in the pandemic before there were alternative tests available.
Tornus40

Thank you. This helped me think more clearly about something we do often.

Answer by Tornus60

Zvi's Omicron summary is probably your best source of information:

Omicron probably milder than Delta (~50%) so baseline IFR likely ~0.3% unless hospitals overload, lower for vaccinated or reinfected.

Answer by Tornus10

Good questions—thank you for starting this conversation.

Your assumptions about testing seem reasonable, and hopefully we'll have confirmatory data soon.

I have with great regret stopped using microCOVID. A factor of 2 - 3 x risk multiplier seems reasonable, but I no longer entirely trust their transmission model. It's probably still more or less valid, but Omicron is a very different disease. There's some interesting data about it preferring the upper respiratory tract to the lungs, and about how Omicron particles behave differently in aerosols, that make m... (read more)

Tornus20

Excellent question!

My best guess: for detecting people who are infectious but asymptomatic, antigen tests will likely perform approximately as well with Omicron as they have with Delta. Because Omicron infections ramp up so fast, however, I'm reducing my guess for how long you can trust the results from 12 hours to 6 hours. (That is to say, if you tested negative this morning, you shouldn't assume that you aren't infectious this evening).

In addition to the data you cite, Abbott claims their testing shows no decrease in BinaxNOW effectiveness against Omicron. That would also be my prior. I'm curious what the FDA has found, although they've been coy about releasing details. I assume we'll see more data soon.

Tornus10

We could certainly have done much better (both before and during the pandemic), but unfortunately it isn't as simple as just giving IBM $100M. Any solution needs to fit into the vast array of other existing systems used for reporting lab results, managing medical records in hospitals, etc.

The US delivers health care in a very patchwork way, which has made the deployment of electronic medical records very slow and difficult. Strong, smart leadership at the top would help a great deal, but even in the best possible case, really fixing this problem would take many years.

Tornus75

microCOVID has been a game changer for me and many people around me: the ability to get quantitative risk assessments radically improved our ability to efficiently spend risk. We recently stopped using it because of Omicron, and I'm very sad about it.

To me, one of the coolest things about microCOVID has been the proof of concept that a group of smart civilians can put together a useful tool that significantly shifts the efficient frontier for navigating Covid. That alone seems valuable to me, and I'd love to see the project keep going as a testbed for how ... (read more)

Tornus*150

I can't speak to San Francisco specifically. But if it's anything like many other locations in the US, the problem isn't malice or indifference: it's that generating this data is vastly harder than you realize. The politicians get the data the same time you do: as soon as it's ready.

Here's one tiny true example, from one part of the pipeline in one particular location. A substantial amount of data enters the system as faxes. The faxes go to a room full of National Guard, who manually enter the data into computers, from whence it begins a complicated proces... (read more)

1knite
I understand that data collection is difficult and empathize with the people responsible for doing the work. The thing is, SF used to publish everything as soon as they could! We accepted that numbers could be revised up or down as data was fully coded. This 5 day lag is IMO far on the wrong side of timeliness vs correctness.
Answer by Tornus50

Rapid antigen tests at the door reduce risk by about 75%, assuming people are asymptomatic and you test each day if it's a multi-day event. I did a deep dive on antigen tests recently, if you'd like to see the data.

PCR is probably similar: they're much more accurate, but the data is more stale, which especially with Delta is a significant issue.

2mingyuan
Thank you! This answers my question :)
Tornus20

That is precisely the question, and I confess that I don't know the answer for certain. I think, though, that both factors are important.

The issue you're talking about is definitely a thing: influenza evolves rapidly enough that any given vaccine will become less effective over time simply because the dominant strain of the virus has drifted.

However, I believe it is also the case that the immune response drops off fairly quickly. I haven't found a definitive source (I confess that I didn't look hard), but the closest I came is this article, with this quote... (read more)

3dawangy
I may have found the answer to my lazy question on the CDC website: https://www.cdc.gov/flu/prevent/misconceptions.htm "Can vaccinating someone twice provide added immunity? In adults, studies have not shown a benefit from getting more than one dose of vaccine during the same influenza season, even among elderly persons with weakened immune systems. Except for children getting vaccinated for the first time, only one dose of flu vaccine is recommended each season." Since they say that "studies have not shown" rather than "we don't have studies that show," I'm more inclined to believe them.
Tornus10

That makes sense—it's also true that the efficacy of the flu shot declines over time (maybe 8% - 10% per month?), so there is significant concern about getting it too early. I could certainly see making an argument for getting one as soon as possible and a booster shot in the mid to late season. That's a single shot with a booster, technically, not a two shot series.

2Zac Hatfield-Dodds
The question is whether that decline in effectiveness is because of declining immune response (in which case an identical booster would help) or a shifting distribution of influenza strains - in which case you'd need a different shot. Of course it's likely to be a mixture, but my understanding is that it's mostly the latter.
Tornus40

Taking the "wait" argument to its logical extreme, it seems to me one could argue not only waiting for kids to get vaccinated, but waiting until COVID rates are minimal, so immune compromised people can safely attend.

I don't think it's necessary or appropriate to take everything to its logical extreme, but it seems to me that if one is going to advocate waiting for one group but not another, it's important to clearly articulate the moral principle behind that distinction.

I'm not a dancer, but my instinct is that a "reasonable accommodation" model is appropriate here: there's a moral imperative to make events as accessible as reasonably possible, but not to cancel any event that isn't 100% accessible to every person.

Answer by Tornus-20

I would doubt it—different vaccines provoke different immune responses, and each has a dosing schedule based on empirical evidence about what produces the best response. The fact that two doses are needed for an optimal response from the covid vaccine doesn't tell you much about any other vaccine.

It's possible that two vaccines would produce a slightly better response but they decided the cost/benefit didn't pencil out, and I could imagine that for some immune compromised people, getting two would be appropriate. But I'd stick to the recommended schedule a... (read more)

6ChristianKl
The dosing schedule often get chosen before the empirical evidence is in. J&J one dose scheduled compared to Moderna's two doses is not based on any empiric justification. The claim that two doses for a COVID vaccine produces an optimal result is also depends a lot on your goals if your goal is not getting infected more then two are better.
Tornus50

Really solid analysis. Regarding rapid tests:

A pretty important downside in many cases is that they're logistically complicated at a large event. The tests need to lie flat on a table or other surface for 15 minutes. Are you gonna have a giant table covered in tests? Do people come in to test and then go back out? Do they take their test out and perform it in their cars? These are solvable problems, but they can add a lot of complexity and crowding to the checkin area, which is already a problem spot at many events.

With that said, I'm a huge fan of tests f... (read more)

2jefftk
They do get you four times as much socializing for the same level of risk, but so do some other options like air purifiers. If you're already doing the others, and you are doing something risky enough or are sufficiently cautious, then rapid tests do make sense. EDIT: this is also different by country; I'm thinking of the US where rapid tests are somewhat hard to find in stock, and cost ~$12/each
Tornus20

OK, did some digging. The relevant source is table 2 from Peng et. al., Practical Indicators for Risk of Airborne Transmission in Shared Indoor Environments and their Application to COVID-19 Outbreaks.

They calculate the following relative risk rates:
Silent: 0.0012 (1x silent rate)
Speaking: 0.0058 (4.98x silent rate)
Shouting / singing: 0.0350 (29.91x silent rate)
Heavy exercise: 0.0817 (69.83x silent rate)

IMHO, you could may argue for a risk factor of 1/10 compared to heavy exercise (which is 7x the silent rate), but my gut is that 1/5 (14x the silent ra... (read more)

Tornus20

If you can afford them, the rapid tests are a great idea: microCOVID doesn't model them, but I believe they cut your risk by about a factor of 4.

2jefftk
I don't think rapid tests are worth it here; see the follow-up.
1Sameerishere
For those who are bad at math words like I am and didn't click through to your link, I want to note that you mean, "they cut risk to 25 percent of the original risk", not "they reduce risk by 25 percent" :-) (I thought you meant the latter till I clicked through)
Tornus20

Yes, I emphatically agree with this (as does my consultant, who is an epidemiologist who works on COVID full time).

I think one can reasonably argue about the details: loud vocalizations create different aerosol patterns than exertion, and off the top of my head I'm not aware of any really solid data on how the two would compare. But I think your numbers are low by at least a factor of 5, and a factor of 25 seems very plausible to me.

Also: you've selected surgical masks when doing the µCoV calculation. Will that actually be true? If most people wear cloth, thin or loose (which seems most typical here), that'll increase the risk by another factor of 4.

2jefftk
On masks: I went with surgical based on my memory that at the outdoor event we held there was a mix of n95, surgical, and well fitting cloth masks. Ex: https://www.jefftk.com/davis-contra-2021-10-13.jpg
Tornus10

Excellent data: thank you! Two things to keep in mind:

  1. The comment on page 5: the study was "Not powered or designed to compare between the groups"
  2. They're only looking at antibody levels (because those are relatively easy to measure), but there's a good argument that some of the differences between strategies will involve activation of B cells & T cells.

See also the limitations on page 33.

1Weekend Editor
Yes, absolutely. The comment on study power is why I was pretty surprised at the FDA and CDC approvals, based on this study which itself says it's underpowered. I think they were mostly motivated by the safety results that boosters were at least as safe as the primers. So if it might do some good and probably does no harm, they can get to an EUA from there. This is not the way they usually behave, but then again, these are not usual times.
Answer by Tornus20

Excellent question, and I think a lot of us are wishing we had more data on this—unfortunately, there is very little data so far. But here's my take:

  1. If you had J & J for your first shot, I think there's enough evidence now to say it's probably (p = 0.7?) better to get Pfizer or Moderna for your booster.
  2. If you had Pfizer / Moderna for your first two shots, my instinct is that J & J might be the better choice, because there's an argument from microbiology that mixing types might produce a more robust response.
  3. If you had Pfizer / Moderna and want
... (read more)
2Weekend Editor
This is definitely true in terms of antibody fold induction: JnJ followed by either Pfizer or Moderna have the highest fold induction ratios. However, they're starting from a lower baseline, since JnJ doesn't induce such high ab levels to begin with. (Though it might be better at training T cells and memory B cells, and have longer persistence? It's kind of frustratingly complicated, to me.) If you look at absolute ab levels, JnJ followed by Moderna looks best. (Data source: FDA presentation by Lyke, cited in previous comment, slide 22.)
Tornus20

Yes, accuracy in antigen tests seems to correlate very strongly with viral load (and presumably therefore with infectivity). This paper found 100% agreement with PCR for Ct 13-19.9 (massive viral load), all the way down to 8% agreement for Ct 30-35.

Ct (cycle time) measures how many amplification cycles were needed to detect nucleic acid. Lower Ct values indicate exponentially more nucleic acid than higher values, although Ct values are not standardized and can't be directly compared between testing facilities.

Tornus20

Thank you for this! I have a few thoughts about antigen tests.

1: I'd recommend the BinaxNOW as the "standard" home antigen test in the US. Broadly speaking it's better studied, more accurate, cheaper, and more widely available than the others. Regarding data...

2: I think the best current source of general data on home antigen tests is this meta analysis from September. The results from multiple papers over the last year have been pretty consistent, but this adds a little more power to the numbers. They come up with:

Overall: sensitivity 68%, specificity 99-... (read more)

3mayleaf
Thanks for linking the meta-analysis and the other papers; will read (and possibly update the post afterwards)! I especially appreciate that the meta-analysis includes studies of BinaxNOW, something I'd been looking for. Nice, I'd been hearing/reading about using cycle count to determine how much a test's results track infectiousness, and that's really to see the results so starkly supporting that. Looking forward to your writeup! 
Tornus30

Yes, those are all excellent points.

I wrote this as a side reference for a deep dive on the BinaxNOW that's coming shortly, and it'll dig into the numerous, complex, and important issues affecting BinaxNOW accuracy. Short version: the accuracy varies substantially, largely based on viral load. And you're correct that repeated tests on the same individual will be strongly correlated.

And you've convinced me to change the example you cite: I'd gone with the first person for narrative consistency, but I'm shifting it to prioritize technical accuracy.